Insulin as a Growth Hormone: The Double-Edged Key to Energy, Vitality, and Disease
Meta Description (SEO):
Insulin is more than blood sugar control—it’s a growth hormone shaping metabolism, vitality, and disease. Discover its hidden role in health and longevity.
Insulin is more than blood sugar control—it’s a growth hormone shaping metabolism, vitality, and disease. Discover its hidden role in health and longevity.
1. Summary / Abstract
Insulin is one of the most misunderstood hormones in the human body. Commonly reduced in public discourse to a “sugar-lowering” agent, insulin is, in reality, a powerful growth hormone that orchestrates energy metabolism, tissue repair, cell proliferation, and survival. Without insulin, life cannot be sustained. With too much insulin, however, modern chronic diseases flourish—obesity, cancer, cardiovascular disease, Alzheimer’s, and autoimmune dysfunctions.
This article dives into insulin’s true multidimensional role: its functions at the cellular, mitochondrial, and systemic levels; its balancing act between growth and destruction; and the holistic approaches—from mainstream endocrinology to Ayurveda and frequency medicine—that can restore insulin harmony. We’ll also address the controversy: how the medical establishment often underplays the dangers of chronic hyperinsulinemia while natural practitioners have warned for decades that insulin, not cholesterol or calories, is the true driver of modern disease.
2. Key Terms & Definitions
- Insulin – A peptide hormone secreted by the pancreas that regulates glucose uptake and acts as a master anabolic (growth-promoting) hormone.
- Insulin resistance (IR) – A state where cells respond poorly to insulin, forcing the pancreas to produce more insulin, leading to chronic hyperinsulinemia.
- Anabolic hormone – A hormone that stimulates cell growth, protein synthesis, and energy storage.
- Glucose uptake – The process of transporting glucose into cells, primarily muscle, liver, and fat tissue, mediated by insulin.
- Mitochondrial redox – The balance of reduction-oxidation reactions within mitochondria that drive ATP production and cellular vitality.
- Hyperinsulinemia – Chronically elevated insulin levels, often preceding type 2 diabetes, obesity, and cancer.
- Hypoglycemia – Low blood glucose, often from excess insulin or lack of counter-regulatory hormones.
- Glycation – A damaging process where sugars bind to proteins, DNA, or fats, forming advanced glycation end products (AGEs) linked to aging and inflammation.
3. Background / Context
Insulin was discovered in 1921 by Frederick Banting and Charles Best, revolutionizing medicine and saving millions of lives from type 1 diabetes. The narrative presented to the public is simple: insulin lowers blood sugar. But physiologists have long known its role is far greater—it is an anabolic signal of abundance. When insulin levels rise, the body shifts into growth, repair, and storage. When insulin levels fall, the body taps into stored energy, burns fat, and initiates autophagy (cellular cleanup).
Modern dietary patterns—constant snacking, processed carbohydrates, and sedentary living—have locked humanity into a state of chronic insulin elevation. The body rarely experiences the natural ebb of insulin that evolution designed. As a result, the growth-promoting nature of insulin, once a survival advantage, becomes pathological: tissues overgrow, blood vessels thicken, fat accumulates, and mitochondria suffocate under oxidative stress.
A polarizing perspective emerges: The world does not suffer from a sugar problem—it suffers from an insulin problem. Sugar is only dangerous in the context of dysfunctional insulin signaling. The same sugar can nourish an athlete post-training but will destroy the arteries of a sedentary office worker already drowning in insulin.
4. Core Content
4.1 Insulin and Cellular Growth
At its core, insulin is an anabolic growth hormone. It stimulates cells to take up glucose and amino acids, triggers protein synthesis, and suppresses protein breakdown. For this reason, bodybuilders sometimes misuse insulin alongside anabolic steroids to accelerate muscle growth—a dangerous practice illustrating just how potent insulin is as a growth signal.
This growth function is a double-edged sword. On one hand, insulin supports recovery from injury, growth in children, and muscle repair after exercise. On the other hand, when insulin is chronically elevated due to modern diets, it drives the proliferation of unwanted tissue—visceral fat, arterial plaques, and even cancer cells. Research shows many cancers thrive in hyperinsulinemic states because insulin activates the mTOR and IGF-1 pathways, which are central to cellular growth.
Here lies the paradox: insulin is required for health and vitality, yet it can also feed the very diseases that shorten lifespan. A finely tuned balance—not too high, not too low—determines whether insulin supports regeneration or fuels degeneration.
4.2 Insulin in Mitochondrial Health
Mitochondria—the energy factories of our cells—are profoundly influenced by insulin. When insulin functions properly, mitochondria receive a steady supply of glucose and fatty acids, enabling efficient ATP production. Insulin signaling also affects mitochondrial biogenesis, redox balance, and electron transport chain activity.
When cells become resistant to insulin, glucose uptake falters, mitochondria struggle, and oxidative stress skyrockets. Instead of burning cleanly, energy production becomes “leaky,” spilling free radicals that damage proteins, DNA, and membranes. This is the beginning of a vicious cycle: poor insulin signaling damages mitochondria, and dysfunctional mitochondria worsen insulin resistance.
Mitochondrial medicine emphasizes that electron flow is life. Insulin is one of the master conductors of that flow. Hyperinsulinemia traps mitochondria in a sugar-burning mode, blocking the natural flexibility to burn fat. This metabolic inflexibility is the hallmark of modern chronic disease. Natural medicine traditions—from fasting in Ayurveda to ketogenic states in modern biohacking—restore mitochondrial flexibility by lowering insulin and reawakening fat oxidation.
4.3 Insulin and the Brain
Insulin does not act only on muscles and fat—it also shapes the brain. Neurons possess insulin receptors that regulate neurotransmitter release, learning, and memory. Insulin enhances synaptic plasticity, the basis of cognitive function. In fact, some researchers call Alzheimer’s “type 3 diabetes,” highlighting the link between insulin resistance and neurodegeneration.
In the short term, insulin spikes can sharpen focus because the brain receives abundant glucose. In the long term, however, chronic hyperinsulinemia dulls the brain. Studies show insulin resistance in midlife predicts dementia later on. Glycation end products stiffen neuronal proteins, while vascular changes reduce cerebral blood flow.
Holistic traditions understood this link intuitively. Ayurveda associates excessive sweet intake with “Kapha derangement” leading to mental dullness. TCM warns that Spleen Qi stagnation (linked to carbohydrate excess) clouds the Shen (spirit). Modern neuroscience now validates these ancient warnings: insulin mismanagement damages both mind and spirit.
4.4 Insulin in Cardiovascular Health
Insulin’s growth-promoting properties extend to blood vessels. While acute insulin spikes increase vasodilation and nutrient delivery, chronic hyperinsulinemia thickens arterial walls and promotes smooth muscle cell proliferation—fueling atherosclerosis. Elevated insulin also worsens lipid profiles, raising triglycerides and lowering HDL.
Furthermore, high insulin drives sodium retention in the kidneys, raising blood pressure. This is why metabolic syndrome often comes with hypertension. Conventional medicine treats hypertension with pharmaceuticals; natural medicine sees the deeper issue: the blood vessels are literally overdosing on insulin signals.
The irony is striking. For decades, cholesterol was demonized as the enemy of the heart. Yet, mounting evidence suggests that insulin—not cholesterol—is the true culprit in vascular aging. A high-sugar, high-insulin diet stiffens arteries far more than cholesterol alone ever could.
4.5 Insulin and Hormonal/Endocrine Systems
Insulin is not an isolated hormone; it cross-talks with nearly every other hormone system. High insulin suppresses growth hormone, disrupting muscle repair and fat burning. It influences sex hormones, contributing to estrogen dominance in women and low testosterone in men.
The link between insulin and fertility is well documented in polycystic ovary syndrome (PCOS). Women with insulin resistance often develop ovarian dysfunction, irregular cycles, and infertility. By restoring insulin sensitivity—through diet, herbs, or metformin—ovulation often resumes.
Thyroid function is also tied to insulin. Hypothyroidism worsens insulin resistance, while hyperinsulinemia alters thyroid hormone conversion. From an integrative perspective, insulin is like the “master gear” of the endocrine clock. When it slips, every other cog in the system misaligns.
4.6 Too Much vs Too Little Insulin
- Too little insulin (Type 1 diabetes, advanced Type 2 burnout) leads to catabolism: muscle wasting, weight loss, ketoacidosis, dehydration, and death if untreated. Insulin therapy is lifesaving.
- Too much insulin (the far more common issue) leads to obesity, fatty liver, hypertension, cancer, cognitive decline, and accelerated aging. It is the slow burn that underlies nearly every chronic disease of modern civilization.
The tragedy is that mainstream medicine often intervenes only when blood sugar rises. Yet insulin levels can be pathological for decades before glucose shows abnormalities. Natural practitioners who test fasting insulin and C-peptide see the early warning signs long before standard doctors do.
5. Multidisciplinary Perspectives
Mainstream Medicine
Focuses on diabetes, insulin therapy, and glucose control. However, it tends to underemphasize hyperinsulinemia as a driver of non-diabetic disease.
Naturopathy
Highlights diet and lifestyle: low-glycemic eating, chromium, magnesium, berberine, cinnamon, and bitter melon to improve sensitivity. Emphasizes intermittent fasting and detoxification.
Homeopathy
Uses remedies like Syzygium jambolanum and Uranium nitricum to balance glycemia. While anecdotal, many practitioners report improvements in energy and sugar control.
Ayurveda
Sees insulin imbalance as a Kapha derangement: heaviness, dampness, accumulation. Treatments include bitter herbs (neem, turmeric, fenugreek), fasting, and Panchakarma detox.
TCM
Frames insulin issues as Spleen Qi deficiency with dampness. Treatments include acupuncture on points like ST36 and SP6, bitter herbs, and lifestyle practices to strengthen digestion.
Quantum & Frequency Medicine
Explores insulin receptor resonance, PEMF, scalar fields, and trauma release to restore receptor sensitivity. Emotional trauma stored in the pancreas or metabolic tissues can block energy flow, worsening insulin resistance.
6. What Assists vs What Blocks Insulin Health
Assists:
- Intermittent fasting & circadian eating
- Regular movement (especially resistance training)
- Minerals: magnesium, zinc, chromium
- Adaptogens: ashwagandha, rhodiola
- Mitochondrial supports: CoQ10, alpha-lipoic acid, methylene blue, red light therapy
- Mind-body practices: meditation, breathwork (lower cortisol → better insulin sensitivity)
Blocks:
- Processed carbs and constant snacking
- Sleep deprivation
- EMF exposure and circadian disruption
- Toxins (BPA, pesticides, heavy metals)
- Chronic stress and unresolved trauma
- Sedentary living
7. Practical Protocols / Biohacking Strategies
- Daily Fasting Rhythm: Eat within a 6–10 hour window. Let insulin rest during the night.
- Carb Timing: Save starches for post-exercise windows, when muscles eagerly soak up glucose without much insulin.
- Herbs & Nutraceuticals: Berberine (500mg twice daily), cinnamon, bitter melon, fenugreek, inositol for PCOS.
- Mitochondria Hacks: Morning sunlight, red light panels, grounding, methylene blue microdosing.
- Emotional Reset: Trauma release therapies to free the pancreas and liver from unresolved emotional blocks.
- Frequency Support: PEMF mats, remote biofeedback (e.g., Spooky2, NLS) to retrain insulin receptor signaling.
8. Unique Narrative / Story Element
Consider the paradox: athletes secretly inject insulin to grow muscle, while ordinary people unknowingly overdose on their own insulin through modern diets. Both cases show insulin as the hormone of growth. The difference is context—one is controlled and paired with training, the other is chronic, silent, and destructive.
The uncomfortable truth is this: we are not dying from too much sugar; we are dying from too much insulin.
9. Conclusion & Call-to-Action
Insulin is not the enemy. It is the silent sculptor of life, a hormone that can build or break us depending on balance. Understanding its dual role—as both healer and destroyer—is essential for anyone seeking longevity, vitality, and resilience.
If you suspect insulin may be silently shaping your health, don’t wait for diabetes to appear. Explore a Total Wellness Evaluation at Natoorales, where hidden metabolic imbalances can be revealed and personalized protocols designed—blending science with natural wisdom.
10. References
- DeFronzo RA. Insulin resistance: a multifaceted syndrome responsible for NIDDM, obesity, hypertension, dyslipidemia. Diabetes Care. 1991.
- Kraft JR. Hyperinsulinemia as the earliest biomarker of diabetes. Clinical Chemistry. 1975.
- Taubes G. Why We Get Fat: And What to Do About It. 2011.
- Lustig R. Fat Chance: Beating the Odds Against Sugar, Processed Food, Obesity, and Disease. 2012.
- Ayurvedic texts: Charaka Samhita, Sushruta Samhita.
- Maciocia G. Foundations of Chinese Medicine. Churchill Livingstone, 2005.
- Practitioner manuals in naturopathy and frequency medicine.
Disclaimer: This article is for educational and training purposes only. It is not medical advice. For personalized care, consult your healthcare provider or holistic practitioner.
Insulin as a Growth Hormone: The Double-Edged Key to Energy, Vitality, and Disease
Meta Description (SEO):
Insulin is more than blood sugar control—it’s a growth hormone shaping metabolism, vitality, and disease. Discover its hidden role in health and longevity.
Insulin is more than blood sugar control—it’s a growth hormone shaping metabolism, vitality, and disease. Discover its hidden role in health and longevity.
1. Summary / Abstract
Insulin is one of the most misunderstood hormones in the human body. Commonly reduced in public discourse to a “sugar-lowering” agent, insulin is, in reality, a powerful growth hormone that orchestrates energy metabolism, tissue repair, cell proliferation, and survival. Without insulin, life cannot be sustained. With too much insulin, however, modern chronic diseases flourish—obesity, cancer, cardiovascular disease, Alzheimer’s, and autoimmune dysfunctions.
This article dives into insulin’s true multidimensional role: its functions at the cellular, mitochondrial, and systemic levels; its balancing act between growth and destruction; and the holistic approaches—from mainstream endocrinology to Ayurveda and frequency medicine—that can restore insulin harmony. We’ll also address the controversy: how the medical establishment often underplays the dangers of chronic hyperinsulinemia while natural practitioners have warned for decades that insulin, not cholesterol or calories, is the true driver of modern disease.
2. Key Terms & Definitions
- Insulin – A peptide hormone secreted by the pancreas that regulates glucose uptake and acts as a master anabolic (growth-promoting) hormone.
- Insulin resistance (IR) – A state where cells respond poorly to insulin, forcing the pancreas to produce more insulin, leading to chronic hyperinsulinemia.
- Anabolic hormone – A hormone that stimulates cell growth, protein synthesis, and energy storage.
- Glucose uptake – The process of transporting glucose into cells, primarily muscle, liver, and fat tissue, mediated by insulin.
- Mitochondrial redox – The balance of reduction-oxidation reactions within mitochondria that drive ATP production and cellular vitality.
- Hyperinsulinemia – Chronically elevated insulin levels, often preceding type 2 diabetes, obesity, and cancer.
- Hypoglycemia – Low blood glucose, often from excess insulin or lack of counter-regulatory hormones.
- Glycation – A damaging process where sugars bind to proteins, DNA, or fats, forming advanced glycation end products (AGEs) linked to aging and inflammation.
3. Background / Context
Insulin was discovered in 1921 by Frederick Banting and Charles Best, revolutionizing medicine and saving millions of lives from type 1 diabetes. The narrative presented to the public is simple: insulin lowers blood sugar. But physiologists have long known its role is far greater—it is an anabolic signal of abundance. When insulin levels rise, the body shifts into growth, repair, and storage. When insulin levels fall, the body taps into stored energy, burns fat, and initiates autophagy (cellular cleanup).
Modern dietary patterns—constant snacking, processed carbohydrates, and sedentary living—have locked humanity into a state of chronic insulin elevation. The body rarely experiences the natural ebb of insulin that evolution designed. As a result, the growth-promoting nature of insulin, once a survival advantage, becomes pathological: tissues overgrow, blood vessels thicken, fat accumulates, and mitochondria suffocate under oxidative stress.
A polarizing perspective emerges: The world does not suffer from a sugar problem—it suffers from an insulin problem. Sugar is only dangerous in the context of dysfunctional insulin signaling. The same sugar can nourish an athlete post-training but will destroy the arteries of a sedentary office worker already drowning in insulin.
4. Core Content
4.1 Insulin and Cellular Growth
At its core, insulin is an anabolic growth hormone. It stimulates cells to take up glucose and amino acids, triggers protein synthesis, and suppresses protein breakdown. For this reason, bodybuilders sometimes misuse insulin alongside anabolic steroids to accelerate muscle growth—a dangerous practice illustrating just how potent insulin is as a growth signal.
This growth function is a double-edged sword. On one hand, insulin supports recovery from injury, growth in children, and muscle repair after exercise. On the other hand, when insulin is chronically elevated due to modern diets, it drives the proliferation of unwanted tissue—visceral fat, arterial plaques, and even cancer cells. Research shows many cancers thrive in hyperinsulinemic states because insulin activates the mTOR and IGF-1 pathways, which are central to cellular growth.
Here lies the paradox: insulin is required for health and vitality, yet it can also feed the very diseases that shorten lifespan. A finely tuned balance—not too high, not too low—determines whether insulin supports regeneration or fuels degeneration.
4.2 Insulin in Mitochondrial Health
Mitochondria—the energy factories of our cells—are profoundly influenced by insulin. When insulin functions properly, mitochondria receive a steady supply of glucose and fatty acids, enabling efficient ATP production. Insulin signaling also affects mitochondrial biogenesis, redox balance, and electron transport chain activity.
When cells become resistant to insulin, glucose uptake falters, mitochondria struggle, and oxidative stress skyrockets. Instead of burning cleanly, energy production becomes “leaky,” spilling free radicals that damage proteins, DNA, and membranes. This is the beginning of a vicious cycle: poor insulin signaling damages mitochondria, and dysfunctional mitochondria worsen insulin resistance.
Mitochondrial medicine emphasizes that electron flow is life. Insulin is one of the master conductors of that flow. Hyperinsulinemia traps mitochondria in a sugar-burning mode, blocking the natural flexibility to burn fat. This metabolic inflexibility is the hallmark of modern chronic disease. Natural medicine traditions—from fasting in Ayurveda to ketogenic states in modern biohacking—restore mitochondrial flexibility by lowering insulin and reawakening fat oxidation.
4.3 Insulin and the Brain
Insulin does not act only on muscles and fat—it also shapes the brain. Neurons possess insulin receptors that regulate neurotransmitter release, learning, and memory. Insulin enhances synaptic plasticity, the basis of cognitive function. In fact, some researchers call Alzheimer’s “type 3 diabetes,” highlighting the link between insulin resistance and neurodegeneration.
In the short term, insulin spikes can sharpen focus because the brain receives abundant glucose. In the long term, however, chronic hyperinsulinemia dulls the brain. Studies show insulin resistance in midlife predicts dementia later on. Glycation end products stiffen neuronal proteins, while vascular changes reduce cerebral blood flow.
Holistic traditions understood this link intuitively. Ayurveda associates excessive sweet intake with “Kapha derangement” leading to mental dullness. TCM warns that Spleen Qi stagnation (linked to carbohydrate excess) clouds the Shen (spirit). Modern neuroscience now validates these ancient warnings: insulin mismanagement damages both mind and spirit.
4.4 Insulin in Cardiovascular Health
Insulin’s growth-promoting properties extend to blood vessels. While acute insulin spikes increase vasodilation and nutrient delivery, chronic hyperinsulinemia thickens arterial walls and promotes smooth muscle cell proliferation—fueling atherosclerosis. Elevated insulin also worsens lipid profiles, raising triglycerides and lowering HDL.
Furthermore, high insulin drives sodium retention in the kidneys, raising blood pressure. This is why metabolic syndrome often comes with hypertension. Conventional medicine treats hypertension with pharmaceuticals; natural medicine sees the deeper issue: the blood vessels are literally overdosing on insulin signals.
The irony is striking. For decades, cholesterol was demonized as the enemy of the heart. Yet, mounting evidence suggests that insulin—not cholesterol—is the true culprit in vascular aging. A high-sugar, high-insulin diet stiffens arteries far more than cholesterol alone ever could.
4.5 Insulin and Hormonal/Endocrine Systems
Insulin is not an isolated hormone; it cross-talks with nearly every other hormone system. High insulin suppresses growth hormone, disrupting muscle repair and fat burning. It influences sex hormones, contributing to estrogen dominance in women and low testosterone in men.
The link between insulin and fertility is well documented in polycystic ovary syndrome (PCOS). Women with insulin resistance often develop ovarian dysfunction, irregular cycles, and infertility. By restoring insulin sensitivity—through diet, herbs, or metformin—ovulation often resumes.
Thyroid function is also tied to insulin. Hypothyroidism worsens insulin resistance, while hyperinsulinemia alters thyroid hormone conversion. From an integrative perspective, insulin is like the “master gear” of the endocrine clock. When it slips, every other cog in the system misaligns.
4.6 Too Much vs Too Little Insulin
- Too little insulin (Type 1 diabetes, advanced Type 2 burnout) leads to catabolism: muscle wasting, weight loss, ketoacidosis, dehydration, and death if untreated. Insulin therapy is lifesaving.
- Too much insulin (the far more common issue) leads to obesity, fatty liver, hypertension, cancer, cognitive decline, and accelerated aging. It is the slow burn that underlies nearly every chronic disease of modern civilization.
The tragedy is that mainstream medicine often intervenes only when blood sugar rises. Yet insulin levels can be pathological for decades before glucose shows abnormalities. Natural practitioners who test fasting insulin and C-peptide see the early warning signs long before standard doctors do.
5. Multidisciplinary Perspectives
Mainstream Medicine
Focuses on diabetes, insulin therapy, and glucose control. However, it tends to underemphasize hyperinsulinemia as a driver of non-diabetic disease.
Naturopathy
Highlights diet and lifestyle: low-glycemic eating, chromium, magnesium, berberine, cinnamon, and bitter melon to improve sensitivity. Emphasizes intermittent fasting and detoxification.
Homeopathy
Uses remedies like Syzygium jambolanum and Uranium nitricum to balance glycemia. While anecdotal, many practitioners report improvements in energy and sugar control.
Ayurveda
Sees insulin imbalance as a Kapha derangement: heaviness, dampness, accumulation. Treatments include bitter herbs (neem, turmeric, fenugreek), fasting, and Panchakarma detox.
TCM
Frames insulin issues as Spleen Qi deficiency with dampness. Treatments include acupuncture on points like ST36 and SP6, bitter herbs, and lifestyle practices to strengthen digestion.
Quantum & Frequency Medicine
Explores insulin receptor resonance, PEMF, scalar fields, and trauma release to restore receptor sensitivity. Emotional trauma stored in the pancreas or metabolic tissues can block energy flow, worsening insulin resistance.
6. What Assists vs What Blocks Insulin Health
Assists:
- Intermittent fasting & circadian eating
- Regular movement (especially resistance training)
- Minerals: magnesium, zinc, chromium
- Adaptogens: ashwagandha, rhodiola
- Mitochondrial supports: CoQ10, alpha-lipoic acid, methylene blue, red light therapy
- Mind-body practices: meditation, breathwork (lower cortisol → better insulin sensitivity)
Blocks:
- Processed carbs and constant snacking
- Sleep deprivation
- EMF exposure and circadian disruption
- Toxins (BPA, pesticides, heavy metals)
- Chronic stress and unresolved trauma
- Sedentary living
7. Practical Protocols / Biohacking Strategies
- Daily Fasting Rhythm: Eat within a 6–10 hour window. Let insulin rest during the night.
- Carb Timing: Save starches for post-exercise windows, when muscles eagerly soak up glucose without much insulin.
- Herbs & Nutraceuticals: Berberine (500mg twice daily), cinnamon, bitter melon, fenugreek, inositol for PCOS.
- Mitochondria Hacks: Morning sunlight, red light panels, grounding, methylene blue microdosing.
- Emotional Reset: Trauma release therapies to free the pancreas and liver from unresolved emotional blocks.
- Frequency Support: PEMF mats, remote biofeedback (e.g., Spooky2, NLS) to retrain insulin receptor signaling.
8. Unique Narrative / Story Element
Consider the paradox: athletes secretly inject insulin to grow muscle, while ordinary people unknowingly overdose on their own insulin through modern diets. Both cases show insulin as the hormone of growth. The difference is context—one is controlled and paired with training, the other is chronic, silent, and destructive.
The uncomfortable truth is this: we are not dying from too much sugar; we are dying from too much insulin.
9. Conclusion & Call-to-Action
Insulin is not the enemy. It is the silent sculptor of life, a hormone that can build or break us depending on balance. Understanding its dual role—as both healer and destroyer—is essential for anyone seeking longevity, vitality, and resilience.
If you suspect insulin may be silently shaping your health, don’t wait for diabetes to appear. Explore a Total Wellness Evaluation at Natoorales, where hidden metabolic imbalances can be revealed and personalized protocols designed—blending science with natural wisdom.
10. References
- DeFronzo RA. Insulin resistance: a multifaceted syndrome responsible for NIDDM, obesity, hypertension, dyslipidemia. Diabetes Care. 1991.
- Kraft JR. Hyperinsulinemia as the earliest biomarker of diabetes. Clinical Chemistry. 1975.
- Taubes G. Why We Get Fat: And What to Do About It. 2011.
- Lustig R. Fat Chance: Beating the Odds Against Sugar, Processed Food, Obesity, and Disease. 2012.
- Ayurvedic texts: Charaka Samhita, Sushruta Samhita.
- Maciocia G. Foundations of Chinese Medicine. Churchill Livingstone, 2005.
- Practitioner manuals in naturopathy and frequency medicine.
Disclaimer: This article is for educational and training purposes only. It is not medical advice. For personalized care, consult your healthcare provider or holistic practitioner.
Ian Kain,
Wellness Thrive Designer
Natoorales.com
+52 958 115 2683, WhatsApp
